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Toxic Black Mold and Biological Weapons

T-2 Trichothecene Mycotoxins and Biological Weapons

Biological weapons have been made from T-2 trichothecene mycotoxins. These are the same mycotoxins produced by toxic black mold (Stachybotrys chartarum), which can be found growing in thousands of homes.

The only difference between the mycotoxins in these biological weapons and the mycotoxins found in homes infested with toxic mold is the concentration level.

Several countries have grown toxic mold to harvest concentrated T-2 mycotoxins for biological weapons.

Toxic Black Mold and Yellow Rain

During the Vietnam War, concentrated T-2 trichothecene mycotoxins were released over remote jungle areas in Laos. This caused over 6300 deaths between 1975 and 1981. These biological attacks were dubbed Yellow Rain because of the eyewitness's descriptions of a yellow oily liquid being released from low flying aircraft.

In one case, witnesses reported seeing a cloud of yellow liquid falling from the sky onto a village. When they entered the village they found every person and animal had died. As they left the village they reported feeling severe shortness of breath.

Yellow Rain attacks also occurred in Kampuchea during 1979 to 1981 leading to over 1000 deaths. They were also used by the Soviet Union in Afghanistan from 1979 to 1981 resulting in over 3000 deaths.

Toxic Black Mold in Gulf War Biological Weapons

During the Gulf War in 1991, Iraq reportedly used biological weapons containing T-2 mycotoxins against American soldiers. After an Iraqi missile was detonated over a US military base in Saudi Arabia, the American soldiers reported symptoms that matched those caused by trichothecene mycotoxins. (Toxic Black Mold Symptoms)

Gulf War syndrome may at least partly be caused by exposure to T-2 mycotoxins. Trichothecene mycotoxins were also reportedly used against Iran in the Iran-Iraq war.

Toxic Black Mold History

To read more about how toxic black mold and its mycotoxins have affected humans in the past visit Toxic Black Mold History. /

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